Hadrian’s Wall: Newcastle’s West End

Debate between Chi Onwurah and Mary Glindon

Tuesday 1st February 2022 Westminster Hall

[Chi Onwurah (Newcastle upon Tyne Central) (Lab)]

I beg to move,

That this House has considered Hadrian’s Wall in Newcastle’s West End.

It is a real pleasure to serve under your chairship, Sir Gary. I like to champion Newcastle as the home of the first industrial revolution and as a hub of today’s green industrial revolution. However, there is another facet to our great, vibrant city that is less well known: Roman Newcastle. As a child, one of my favourite shows was “Star Trek”. I loved the phrase, “Space: the final frontier.” I was born in Wallsend, but I did not realise that in Roman times the final frontier was not space, but Newcastle, which marked the northernmost boundary of the Roman empire.

This year, we celebrate Hadrian’s Wall’s 1,900th birthday, and we need to celebrate all the wall. Hadrian’s Wall tends to conjure up images of the wonderful Northumbrian countryside, but the wall is and was an urban wall, too. It runs through the wonderful, vibrant, multicultural, urban west end of Newcastle, but not everyone knows that. Many tourists are actually directed away from the wall by the Hadrian’s Wall National Trails path and other trails and tours that follow the wall, such as those of the Ramblers Association and the National Cycle Network. That is not right. It is not right that the west end of Newcastle should be missed out of our national Roman heritage.

We must remember that the wall was built by an invading and colonising army. Hadrian himself said that it was to keep his empire intact—a duty that he felt was imposed on him by divine instruction. We do not have a record of what the indigenous peoples of the north thought, but the wall must have divided families and communities, as walls that are constructed to keep people out always do. While we celebrate the heritage and history of the wall, we do not celebrate Rome’s hierarchical slave society. I am glad to say that Newcastle does not seek to emulate that particular aspect of our heritage, being a long-standing centre of the struggle for social justice. However, our Roman heritage is deep within us.

Our city was named for the new Norman castle that stands on the site of the Roman fort of Pons Aelius—Aelius was Hadrian’s family name, so it was “Hadrian’s bridge”. We can be relatively sure that some of the stones from the wall were recycled into that castle. Indeed, many buildings—particularly churches, which were the first major stone buildings built after the Roman withdrawal—undoubtedly have stones from Hadrian’s Wall within them.

There are still significant traces of the wall in my constituency. Just last year, 3 metres of some of the oldest parts of the whole wall were found in the city centre during routine drain maintenance. The remains of Milecastle 4 can be found at Newcastle Arts Centre, less than 100 m away from my constituency office, which is also in the city centre. Yet every day, tens of thousands of tourists pass by without knowing how close they are to the Roman wall.

[Mary Glindon (North Tyneside) (Lab)]

My hon. Friend is making a fantastic case for Hadrian’s Wall—not the “Roman wall”; there are others—in the west end of Newcastle. Talking about all of the wall for this 1,900th anniversary is so important. I know that today is about being inclusive of all parts of the wall, so I hope she agrees with the idea developed in Wallsend in my constituency, where I live and she was born. In the planned redevelopment of the Segedunum Roman fort, Tyne & Wear Archives & Museums and North Tyneside Council, are keen to explore redirecting the trail through the 80 metres of wall foundations that were repaired by the Romans and a reconstructed part of the wall that people can climb. Hadrian’s Wall Partnership Board includes in its 10-year investment programme the establishment of stopping points to highlight the wall in unexpected places—

[Sir Gary Streeter (in the Chair)]

Order. I am afraid that interventions must be a lot shorter than that.

Transport: North-east

Debate between Chi Onwurah and Mary Glindon

Wednesday 23rd November 2016 Westminster Hall

[Chi Onwurah]

I thank my hon. Friend, the Chair of the Business, Energy and Industrial Strategy Committee, for making such an important point, which I will come on to properly later. I agree wholeheartedly with the implication of his comments. The economic contribution of effective transport infrastructure for the north-east is not recognised in the same way as it is recognised in London and other areas of the south. That absolutely has to change if we are to have any hope of rebalancing our economy and making it more resilient and distributive across the country.

[Mary Glindon (North Tyneside) (Lab)]

My hon. Friend set out what she will speak about, but airport passenger duty is also important to the economics of the north-east. Our airports play an equally valid and massive role in helping our economy. We heard nothing today about what will happen with regard to Scotland, but it will be detrimental to our north-east airport if the Scottish Government reduce APD.

[Chi Onwurah]

Given the interest in and strategic importance of transport, I will focus on the issues I set out, but my hon. Friend makes a critically important point. Newcastle airport is a vital part of our economic infrastructure. Naturally, it competes with airports in Scotland. The lack of a decision today—I am not sure whether it has been kicked into the long grass or into orbit—is detrimental to economic certainty at a time of great uncertainty for many other aspects of our economic future.

Let me talk briefly about rail and the metro. I was nine or 10 when the metro came into being. It was a fantastic, highly advanced network that was ahead of its time—I think it was the first network in Europe or the world to be accessible to disabled people—but 40 years later we are using exactly the same rolling stock. Is that believable? Hitachi recently told me that it can deliver trains that would provide what we have been talking about—an extended light rail and metro service across a greater part of Tyne and Wear and the north-east. I hope the Minister will commit to that investment, because we need transport infrastructure and a metro without delays to support the kind of economy we want.

Most of Newcastle Central’s transport is about buses. We have a number of metro stops, but for most of the west of my constituency and parts of the north it is about buses. The failure of bus deregulation in Tyne and Wear has been so patently obvious for so many decades that it beggars belief that we are still debating it today. Outside my constituency office near Central station in Newcastle two No. 1 buses leave in totally different directions, one going north, one south—they both have the same number, because obviously that puts them at the head of some queue. It is totally incomprehensible to those who have lived in the city for many years, never mind visitors.

As my hon. Friend the Member for Houghton and Sunderland South discussed, we should not still be debating the lack of integrated bus transport in 2016, when we have seen the success of, for example, the Oyster card and the integrated system in London. I really cannot believe that the Minister will stand up to say that Tyne and Wear and my constituents do not deserve some control over a bus system that is so important to them simply because of the lack of a mayor.

Why is this all so important? As my hon. Friends the Members for Hartlepool (Mr Wright) and for Houghton and Sunderland South said, transport is important because it is part of our economic infrastructure and the north-east having critical mass.

Severe Eating Disorders (North-East England)

Debate between Chi Onwurah and Mary Glindon

Monday 23rd June 2014 Commons Chamber

[Chi Onwurah]

The hon. Gentleman makes an excellent point, and I certainly believe that the decision should and must be reviewed. It is clear that a number of hon. Members have been contacted by concerned constituents. Indeed, the right hon. Member for Berwick-upon-Tweed (Sir Alan Beith), who cannot attend this debate, asked me to say that he also had constituents who are affected.

Given that admissions in the north-east are 30% above the national average, and that the Royal College of Psychiatrists recommends that six beds per million of the population are needed for average admission rates, the north-east’s 2.8 million people need 23 beds. I will return to that figure, but first a word about the threatened unit that hon. Members have already referred to.

The Richardson eating disorder service is operated by Northumberland, Tyne and Wear NHS Foundation Trust. It is in the centre of Newcastle, with excellent transport links. It is acknowledged to be an outstanding unit, rated excellent by the Royal College of Psychiatrists and the Care Quality Commission. It has just won Beat’s clinical team of the year award. A stable, vastly experienced staff has been treating adult in and out-patients since 1997, and it has saved many lives. One sufferer said:

“I have suffered from anorexia nervosa for over 12 years and unfortunately during that time I have required many admissions to medical and eating disorder units”.

She names a number of them before going on to say:

“The admission to the Richardson was by far the most successful. I made such huge strides towards recovery and was the healthiest I have been since this all began.”

[Mrs Mary Glindon (North Tyneside) (Lab)]

In the light of what my hon. Friend says, and the fact that the unit’s model of partial hospitalisation of out-patient services has been so successful, does she not agree that NHS England should look at that model and perhaps reconsider its decision on the specification of specialised services?

[Chi Onwurah]

My hon. Friend makes an excellent point. Indeed, if the criteria on which this decision was made were publicly available, we could perhaps tell which models NHS England considered and what it hoped to achieve. Unfortunately, there is no transparency, which is one of the key issues.

Problems started in 2010, when commissioned adult eating disorder in-patient beds were tendered and the contract was awarded to Tees, Esk and Wear Valleys NHS Foundation Trust, although it did not then operate an in-patient unit. It quickly established a 10-bed unit in Darlington, but on a site with poor transport links to the north. For clarity for those Members who may not be familiar with the north, Newcastle is to the north of Darlington.

The award was a shock to many people, not simply because of the result, but because of the lack of consultation. I should like to ask the Minister a specific question: against what criteria were proposed services considered to be better than award-winning ones already on offer in the Richardson? If he does not know, I hope that he will promise to find out. Was cost the driving factor? What was the evidence basis for the centralising of these critical mental health services?

The National Institute for Health and Care Excellence guidelines specifically state that for severe eating disorders patients should be treated near their homes, with the support of family and friends. These are often young, vulnerable people, who are not yet independent of their family, either financially or emotionally. As one told me,

“Seriously ill anorexics are often cognitively impaired as a result of severe starvation and separation from loving support, together with that the challenge to dangerous and entrenched behavioural traits is often too much to bear.”

Given the lack of consultation, the north-east specialised commissioning group was instructed to strengthen its relationships with stakeholders and report any other substantial changes or developments to the NHS scrutiny committee.

NEEDAG, formed by carers and patients concerned about the threat to the Richardson, hoped that at least five of the beds in the Richardson would continue to be used by those in the north of the region, given overall regional demand. However, in April 2012, the commissioner increased the number of beds at Darlington to 15—again, without any consultation, scrutiny or performance data by which to make judgments. When challenged, I am told that the commissioner said they were not obligated to consult anyone. I hope that the Minister will correct them on this point. It is possible that the top-down reorganisation of the NHS instituted by this Government may have led to them forgetting their obligations under the NHS constitution.

When Darlington was full, commissioners started sending very ill patients out of the area, instead of to the Richardson, saying that every commissioned bed in England, no matter where it was, had to be filled before a patient from Tyneside could be sent to Newcastle. That is how we have arrived at the ridiculous and tragic situation of our national health service sending vulnerable Tyneside patients to Glasgow, Norwich and London when there are empty beds in the Richardson unit in the centre of Newcastle.

The impact on vulnerable young people of being separated from their families undoubtedly makes it more difficult to recover—hence the NICE guidelines. The cost of visiting for families is enormous, both financially and emotionally. One parent wrote:

“This will then have an effect on our family’s mental health as we are all struggling to come to terms with the condition and to help M recover. I would refuse to let M be admitted so far away from home and would rather give up my full time job to look after her in the familiar and safe surroundings of home.”

Another parent who fought to win a place for their daughter at the Richardson said:

“We were very angry to have been put in the position of having to fight for a bed for our dangerously ill daughter at a time when all our energy was needed to comfort and support her through a very difficult time. The added pressure and anxiety it caused the whole family was dreadful.”

It has been announced that the unit will be closed down, because it was said—cynically and cruelly—that it was not being used locally. If it was not being used locally, it was because NHS England was sending local people hundreds of miles away. Freedom of information requests submitted by NEEDAG show that Darlington’s 15 beds are full; that there are eight in-patients from the north-east in London, Sheffield, Leeds, Glasgow and Norwich; and that five patients have managed to win beds in the Richardson.

Mitochondrial Disease

Debate between Chi Onwurah and Mary Glindon

Tuesday 25th June 2013 Westminster Hall

[Chi Onwurah (Newcastle upon Tyne Central) (Lab)]

It is a great pleasure to serve under your chairmanship, Mr Davies. I am pleased to be able to lead a debate on this most important subject. I shall speak about mitochondrial disease, the devastation it causes and the new techniques developed by Newcastle university to prevent it. I declare an interest in that my father studied medicine at Newcastle, so I am a natural champion of that great university’s medical research and innovation. I am here primarily to champion not Newcastle university however, but the interests of my constituents struck down by mitochondrial disease, and indeed all those who suffer from it.

The subject is technical and I will attempt to be as clear as possible in setting out the arguments. Mitochondria are found in every cell in the human body, except red blood cells. They are the batteries generating energy for the cell. Mitochondria convert the energy of food molecules into the energy that powers the cell’s functions. About 200 children are born every year with a mitochondrial disease. Such diseases are passed from mothers to their children and are caused by faulty mitochondria. Like all DNA, the DNA in mitochondria can mutate and mothers can pass those mutations on to their children. Faulty mitochondria mean that the cells are unable to function normally and the diseases caused by them can have a devastating effect on families. The diseases tend to affect parts of the body that use a lot of energy, such as the brain, muscles, nerves, liver, kidney and heart, and vary widely in severity, from life-threatening to having few or no obvious symptoms. Symptoms vary, but can include poor growth, muscle weakness, tiredness, poor co-ordination, and sensory, respiratory or cognitive problems.

There are no effective treatments available for serious mitochondrial disease. When the cells go wrong, it can result in serious conditions, including blindness, fatal heart failure, liver failure, learning disabilities and diabetes, and can lead to death in early infancy. Prevention is the only realistic option. In 2010, Newcastle university scientists, with funding from the Wellcome Trust, pioneered research into variations of in vitro fertilisation procedures that could prevent the transmission of the genetic mutations that cause these devastating disorders. The techniques use part of an egg donated by a healthy individual, to replace the faulty mitochondria of the affected mother. The intention is to give affected families a chance to have healthy children that are genetically related to them, but born free of mitochondrial disorders. Such techniques are not currently permitted in the UK, but legislation allows the Government to introduce secondary legislation that would allow the treatments to be used.

Mitochondrial disease can blight families for generations, because, as I said, it is passed from the mother to child during pregnancy. The techniques could put a stop to it, by preventing the faulty mitochondria from being passed to the embryo. Mitochondrial disease affects about 6,000 adults in the UK. In my constituency, four families—Bumstead, Cass, Bland and Mahmood—suffer from mitochondrial disease. Although every effort is being made to help them, there is no cure. For example, Lily Cass, who is in her 70s now, has five brothers and three sisters, and one brother who died at 56. They are all affected in different ways by mitochondrial diseases, and some more severely than others. Some days, Lily can hardly move due to lack of energy caused by her faulty mitochondria, which takes all her strength away. She has four children, including a daughter, who is likely to pass the disease on to her children. She worries about that all the time.

For those women and their families, the most important help we can offer is potential treatments, to prevent the next generation of patients from being affected. The opportunity to have their own children free of disease is something that the patients understandably want.

As with all such advances, it is right that the ethics are properly considered before techniques are adopted, and the Minister will be aware that concerns have been raised. There are those who argue that the techniques create children with three parents, but the embryo would carry only a small number of genes from the donor—just 13 out of 23,000, or 0.056% of the genetic material. How much of a parent is that? The function of the 13 genes is restricted to powering the mitochondria; they do not affect personal characteristics such as eye or hair colour, or behaviour.

Last June, the Nuffield Council on Bioethics produced a report that found that the technique would be an ethical treatment option for affected families, as long as research showed that treatment was likely to be safe and effective, and families were offered full information and support. The council’s report found that no strong cultural or social emphasis is generally placed on mitochondrial inheritance as a specific element of personal identity. Many of the social and biological aspects that typically imply a “parent”, and may be relevant in egg donation for reproduction, do not apply to mitochondrial donation. The council therefore suggested that if the treatments were made available, mitochondrial donors should not have the same status in regulation as reproductive egg donors.

[Mrs Mary Glindon (North Tyneside) (Lab)]

My hon. Friend makes a good case about the serious effects that the diseases resulting from the condition have on families. If we think forward to any children who are fortunate enough to be born without disease because of the treatment, would there be any possibility that they might consider themselves to have three parents, whether or not they had any traits from the third one? Has thought been given to how that would be considered if it should happen?

[Chi Onwurah]

My hon. Friend is right. There has been some debate about the status not only of the donors but, most importantly, of the children. The Nuffield Council on Bioethics says that families must be offered full information and support, and that must also apply to the children, so that they understand the scientific nature of the very limited gene inheritance from the donation.

If mitochondrial donors were not given the same status as reproductive egg donors, it would be not legally required for them to be identifiable to people born from their donations. The council concluded that the proposed treatments would be a form of gene therapy that would permanently cure the disease in future generations. Changes resulting from the replacement of mitochondrial DNA would be passed on not only to the resulting children, but to the descendants of any girls born from the techniques, via their eggs.

Dr Geoff Watts, who chaired the inquiry, said:

“We understand that some people concerned about the idea of germline therapies may fear that if such treatments for mitochondrial gene disorders were approved, a ‘slippery slope’ would be created towards comparable alterations to the nuclear genome.”

That is an understandable fear, but he went on to make a very important point:

“However, we are only talking about the use of these techniques in the clearly-defined situation of otherwise incurable mitochondrial disorders, under strict regulation.”

In 2012, the Human Fertilisation and Embryology Authority—HFEA—launched a public consultation on mitochondria replacement. It interviewed almost 1,000 people, and a further 1,800 completed questionnaires. It also organised public workshops around the UK and spoke to individuals affected by the diseases, to gauge their views. It published the results in March of this year, and found broad public support for the use of the technique.

The HFEA asked four main questions about attitudes to the gene treatment of mitochondrial diseases. When asked about attitudes to the selection of embryos based on testing, 65% of those questioned were positive or very positive, with only 8% negative. When asked about altering the genetic make-up of an egg or an embryo, 56% were positive or very positive and only 10% were negative. Attitudes to the use of genetic material from a third person showed that 44% were positive or very positive, with only 15% negative. The HFEA therefore advised the Government that there was broad support for mitochondrial replacement being made available to families at risk of passing on a serious mitochondrial disease. It also advised that if treatment were to be authorised by Parliament, it should be under certain conditions, such as its being available only in licensed clinics.

The HFEA recommendations have been widely welcomed by campaigners. For example, Dr Marita Pohlschmidt, director of research at the Muscular Dystrophy Campaign, said:

“We welcome this outcome. There is currently no effective treatment available for mitochondrial diseases, and at this time, prevention remains our strongest option. By taking forward research into pro nuclear IVF, we move towards giving women living with these devastating and unpredictable conditions the choice to bear their own unaffected children. This technique does involve a step into new scientific territory. But it is a calculated, specific step with the sole aim of preventing a potential fatal condition from being passed down to the next generation, where possible.”

We are now waiting for a decision from the Government about whether secondary legislation that will allow the techniques to be licensed for use in patients will be introduced in this parliamentary Session. It has taken years to get to this stage, and it is important that progress does not stall because families are waiting for this. Introducing regulations now will ensure there is no avoidable delay in the treatments reaching affected families once research is completed and the HFEA considers there to be sufficient evidence that the techniques are safe and effective.

I called this debate to hear an update from the Minister on the progress that she has made, and to ask when we can expect a decision, and when we can expect to see legislation.